Which condition would not contribute to secondary hypertension?

Secondary hypertension is elevated blood pressure that results from an identifiable and specific cause. Conditions that lead to secondary hypertension include hormonal imbalances, structural abnormalities, and certain diseases that affect the kidneys or vascular system.

Hyperlipidemia, which refers to elevated levels of lipids in the blood, does not have a direct causal relationship with hypertension. While it can contribute to cardiovascular disease over time and is often associated with lifestyle factors that may also lead to hypertension, it is not considered a direct cause of increased blood pressure. In contrast, the other options listed—thyroid disease, coarctation of the aorta, and elevated aldosterone levels—are well-recognized conditions that can cause secondary hypertension through their specific physiological effects on blood pressure regulation.

Thyroid disease can lead to hypertension through mechanisms related to thyroid hormone effects on the cardiovascular system. Coarctation of the aorta causes an increase in blood pressure due to the differential pressures above and below the narrowed segment. Elevated aldosterone levels can cause hypertension through sodium retention and increased blood volume. Each of these conditions contributes directly to the development of secondary hypertension, making hyperlipidemia the unique option that does not contribute in the same manner.